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Notably, acquired G6PD deficiencyinduced, for instance, using the inhibitor aldosteroneis associated with NOS uncoupling as well, leading to the increased formation of superoxide anion and peroxynitrite despite the reduction of NO [31]

6o), the contributions to the maximal OCR value in each genotype by ATP-linked OCR , proton leakage, reserve capacity, and non-mitochondrial OCR were altered by Gclc deficiency (Fig

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doi: 10.3389/fnut.2025.1494351 309 ZengLYangTYangKYuGLiJXiangWet al