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Although it is well known that GLP-1R agonists inhibit food intake and reduce body weight, Glp-1r / mice are protected from high fat diet-induced obesity and insulin resistance and have decreased hepatosteatosis [16][18]

Expert consensus on personalized initiation of glucose-lowering therapy in adults with newly diagnosed type 2 diabetes without clinical cardiovascular disease or chronic kidney disease

Glucagon receptor activation appears counterintuitive in obesity and diabetes therapy because glucagon acutely increases hepatic glucose output

[22] reviewed the characteristics of the nasal delivery devices in general and the aerosol generation to achieve the clinical target of nasal drug delivery to the nose cavity, whether the substance in question has local action, is intended for systemic absorption, N-to-B transport, or a combination [22]

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