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This copper peptide gene regulation manifests in several clinically significant ways: Downregulation of Pro-Inflammatory Genes: It actively suppresses genes associated with inflammation, such as those in the IL-6 and NF-B pathways, which are major drivers of cellular aging

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Viral replication and cell to cell transmission suppress ACE2 expression, which has an essential role in the renin angiotensin aldosterone system (RAAS), acting as a key counter-regulatory component