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10.2147/JIR.S350109 469 WangQ.LuoY.Ray ChaudhuriK.ReynoldsR.TanE.-K.PetterssonS

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The numerous CF animal models that develop airway disease similar to humans [162,163,164,165], as well as the multiple small molecules available that inhibit or potentiate NPY signaling [182, 189] offer ample opportunities to investigate the potential role of NPY in CF