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Apart from their role in insulin secretion, GLP-1RAs delay gastric emptying, stimulate the appetite centers in the brain to induce early satiety, and consequently reduce food consumption [13, 26, 56]

Observational data examining hemoglobin trends in patients receiving GLP-1 therapy remain limited and often confounded by comorbid conditions

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Hepatic triglycerides decreased in response to carnitine supplementation possibly due to increased fatty acid oxidation

7 The evolving public perception of obesity and food addiction As the success of GLP-1RAs in treating overweight and obesity rises, it prompts reflection on how these pharmacological advances could reshape both public and professional perspectives