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Inducible expression of the g-glutamylcysteine synthetase light subunit by t-butylhydroquinone in HepG2 cells is not dependent on an antioxidant-responsive element

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Conversely, VTA DA neurons, which are less vulnerable than SNpc DA neurons, generate broad spikes and are autonomous pacemakers, but have smaller Cav1 channel currents and a strong intrinsic calcium buffering by calbindin

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