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However, the ACSL4 protein expression levels were markedly decreased and the GPX4 protein expression levels were markedly increased in the RGSH group compared with the AKI model group (Fig

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found that rIPC may attenuate mitochondrial dysfunction in AKI by inhibiting NOX4-ROS signaling (Long et al., 2022), rIPC protected kidney function and pathological injury and mitigated NADPH oxidase 4 (NOX4) upregulation in different AKI models (cisplatin, LPS and IRI)