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With a deeper understanding of ferroptosis during the occurrence and progression of cerebral I/R injury, new therapies to treat ischemic nerve damage by interfering with the molecular mechanism of ferroptosis are expected to be realized to some extent

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Table S4), presumably because the two cysteines (C20 and C201) remain cappable by HPE-IAM only for these samples

The problem is that peptides are often marketed as low-risk solutions, which can cause patients to overlook potential side effects