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Automated image analysis of the distribution of lysosomes marked by LAMP2 demonstrated that catalytically active wildtype TBC1D9B but not the inactive GAP mutant could rescue lysosome dispersion induced by loss of TBC1D9B (Fig

Its used to treat macular degeneration, liver inflammation, fluoroquinolone toxicity and numerous other symptoms and conditions

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The central mechanism of action differentiates the compound from peripherally acting vasodilatory agents and has generated significant research interest in models of CNS-regulated physiological responses

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