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Furthermore, impaired communication between the ER and mitochondria-especially through mitochondria-associated membranes (MAMs)-appears to exacerbate these stress responses, reinforcing the interplay between calcium dysregulation, redox imbalance, and cell death

Munir et al., 1994b)

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A number of small datasets have suggested a potential role for skewed X chromosome activation (XCI), away from the expected 50:50 parent of origin ratio, as an explanation for the strong female preponderance seen in the common autoimmune thyroid diseases (AITD), Graves disease (GD) and Hashimotos thyroiditis (HT)

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