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The extrinsic pathway is driven by extracellular or membrane-associated events, including increased iron uptake via transferrin receptor 1 (TFR1) or inhibition of the xc-cystine/glutamate antiporter system, leading to cysteine depletion and impaired glutathione synthesis

Ferroptosis, an atypical type of cell death first reported by Dixon and colleagues in 2012 (2), is fundamentally different from other types of cell death, such as pyroptosis, apoptosis, autophagy, and necroptosis (3, 4)

GHK-Cu mechanism and discontinuation implications GHK-Cu functions through multiple mechanisms creating skin improvements, with discontinuation effects depending critically on whether peptide presence remains necessary for maintaining induced changes or whether improvements become self-sustaining after initial stimulation period

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