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By reducing peripheral inflammatory signals, improving vascular health, and restoring metabolic homeostasis, semaglutide may alleviate the major drivers of neurodegeneration, suggesting that its neuroprotective benefits may not depend solely on high concentrations within the brain parenchyma

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This finding underscores the conformational plasticity of GPCRs and the limitations of in silico approaches for identifying novel allosteric sites on class B receptors