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Hot flashes result from declining estrogen levels affecting the hypothalamic thermoregulatory center, which narrows the thermoneutral zone and makes the body more susceptible to triggering inappropriate cooling mechanisms like sweating and vasodilation in response to normal temperature fluctuations

Thymosin 4 promotes endothelial progenitor cell angiogenesis via VEGF-dependent mechanism shows T4s angiogenic effect through VEGF and progenitor cell pathways

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ROS can act as secondary messengers in intracellular signaling cascades, affecting processes such as cell proliferation, apoptosis, and differentiation [103]