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[2] Research suggests BPC157 may function through multiple, overlapping intracellular signaling pathways, engaging growth factor receptor networks, nitric oxide biosynthesis cascades, cytoskeletal regulatory mechanisms, and neurotransmitter-modulatory systems

Cell culture studies provide explanations of PBM-induced anti-apoptosis based on identified protein modifications decreasing an apoptosis-regulating protein and suppressing the activity of components for efficient apoptosis [56]

In conclusion, LAbs@Lip@BMn/Rapa exhibited remarkable neuroprotective effects by effectively alleviating the pathogenic neuroinflammation and oxidative stress associated with AD

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