Thus, under asthmatic conditions, intracellular GSH levels could reflect not only the generation of reactive hydroperoxy-phospholipids by the 15LO1-PEBP1 complex, but also the compensatory levels and activity of GPX4, the availability of newly synthesized and regenerated GSH, as well as extracellular transport
An increase in GPX4, specific to dystrophic axons, could explain the apparent increase in GPX4 in putamen, which may be masked by a lack of change in cell bodies of neurons intrinsic to putamen
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Examination of some of the specific signs is technically possible in mice, but has not been done in Lurchers, for example, gaze instability (described in hot-foot mice [209]) or vocalization recording and analysis (however, the question is whether analogy to dysarthria sensu stricto can be seen in mice, because of the substantially different vocalization patter in mice and humans)
