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In the HK-2 cell model transfected with KCa3.1 siRNA and cultured in a transforming growth factor-beta 1 (TGF-1) environment, as well as in the STZ-induced KCa3.1+/+and KCa3.1-/- mouse models, it was found that calcium-activated potassium channel (KCa3.1) deficiency can restore abnormal mitophagy [120]

Kalhan SC, Gruca LL, Parimi PS, OBrien A, Dierker L, Burkett E

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