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OTA was able to increase cellular oxidative stress and apoptosis rates by activating the phosphatidylinositol 3-kinase/threonine kinase (PI3K/AKT) signaling pathway to hinder cell proliferation and development [80]
Neurological Research, 46 (2), 165-177
Tat-induced oxidative stress, DNA damage, and chromosomal aberrations are novel oncogenic factors promoting B-cell lymphomas in HIV-1 infection 123
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