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In apoptosis, -sitosterol upregulates pro-apoptotic factors such as p53 and Bax, while downregulating anti-apoptotic proteins such as Bcl-2, leading to an increase in mitochondrial outer membrane permeability and the activation of key apoptotic enzyme cascades such as caspase-9 and 3, which induces programmed cell death (83)

Jurkat cells were originally isolated almost 40 years ago from an Epstein Barr virus-negative, non-Hodgkins lymphoblastic leukemia 25 , followed by immortalization as a cell line, and, since then, have been widely used for studying signal transduction cascades and cell adhesion 26,27,28,29

Glutaredoxin 2 prevents aggregation of mutant SOD1 in mitochondria and abolishes its toxicity

The real bottleneck in brain peptides is no longer the science

Targeting the tumour vasculature: insights from physiological angiogenesis